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L-(+)-Arabinose

CAS No. 5328-37-0

L-(+)-Arabinose ( —— )

产品货号. M26571 CAS No. 5328-37-0

L-(+)-阿拉伯糖抑制肠道蔗糖酶活性,从而减少蔗糖利用率,从而减少脂肪生成。

纯度: >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
规格 价格/人民币 库存 数量
50MG ¥140 有现货
500MG 获取报价 有现货
1G 获取报价 有现货
1 mL x 10 mM in DMSO ¥124 有现货

生物学信息

  • 产品名称
    L-(+)-Arabinose
  • 注意事项
    本公司产品仅用于科研实验,不得用于人体或动物的临床与诊断
  • 产品简述
    L-(+)-阿拉伯糖抑制肠道蔗糖酶活性,从而减少蔗糖利用率,从而减少脂肪生成。
  • 产品描述
    L-(+)-Arabinose inhibits intestinal sucrase activity, thereby reducing sucrose utilization, and consequently decreasing lipogenesis.(In Vitro):L-(+)-Arabinose is found to be associated with ribose-5-phosphate isomerase deficiency, which is an inborn error of metabolism.L-(+)-Arabinose selectively inhibits intestinal sucrase activity in a noncompetitive manner and suppresses the plasma glucose increase due to sucrose ingestion.?(In Vivo):L-(+)-Arabinose feeding reduced the weights of epididymal adipose tissue in rats. Moreover, plasma insulin and triacylglycerol concentrations were significantly reduced by dietary L-(+)-Arabinose. L-(+)-Arabinose suppresses gluconeogenesis through modulating AMP-activated protein kinase in metabolic disorder mice.
  • 体外实验
    L-(+)-Arabinose selectively inhibits intestinal sucrase activity in a noncompetitive manner and suppresses the plasma glucose increase due to sucrose ingestion. L-(+)-Arabinose is found to be associated with ribose-5-phosphate isomerase deficiency, which is an inborn error of metabolism.
  • 体内实验
    ——
  • 同义词
    ——
  • 通路
    Proteasome/Ubiquitin
  • 靶点
    Endogenous Metabolite
  • 受体
    Interleukin
  • 研究领域
    ——
  • 适应症
    ——

化学信息

  • CAS Number
    5328-37-0
  • 分子量
    150.13
  • 分子式
    C5H10O5
  • 纯度
    >98% (HPLC)
  • 溶解度
    In Vitro:?H2O : 500 mg/mL (3330.45 mM)
  • SMILES
    OC[C@H](O)[C@H](O)[C@@H](O)C=O
  • 化学全称
    ——

运输与储存

  • 储存条件
    (-20℃)
  • 运输条件
    With Ice Pack
  • 稳定性
    ≥ 2 years

参考文献

1.Shimonkevitz R, et al. A diketopiperazine fragment of human serum albumin modulates T-lymphocyte cytokine production through rap1. J Trauma. 2008 Jan;64(1):35-41.
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